Subacromial Impingement

Specialized surgical treatment by Dr. Stéphane Vasseur

Subacromial Impingement

Subacromial impingement corresponds to excessive and abnormal friction between the rotator cuff tendons and the acromion. This frequent pathology requires specialized management to avoid evolution towards tendon lesions.

Subacromial impingement anatomy - Detailed medical illustration
Mechanism of subacromial impingement and tendon friction

What is subacromial impingement?

Subacromial impingement corresponds to excessive and abnormal friction between the rotator cuff tendons and the acromion (acromial bone). By extension, friction can also occur with the outer quarter of the clavicle or the coraco-acromial ligament located in the extension of the acromion.

The manifestations of this subacromial impingement most often occur during anterior or lateral arm elevation movements. Chronic irritation and friction of the tendons within the subacromial space can lead to tendon lesions or bursitis phenomena.

Types of subacromial impingement

Bony conflict

Primary conflict (bony)

In this case, it is the shape of the subacromial vault (aggressive) that will directly come to rub against the rotator cuff tendons.

The shape of the acromial bone is classified into three categories according to Bigliani:

  • Type I: Flat acromion (~17%)
  • Type II: Curved acromion (~43%)
  • Type III: Hooked acromion (~40%) — the most conflict-producing
Muscular conflict

Secondary conflict (muscular)

In this case, subacromial impingement comes from muscular deficiency. There is a desynchronization of the different shoulder muscles, preventing normal mobilization and physiological sliding between tendons and subacromial bone.

Most often there is an anterior displacement as well as an elevation of the shoulder within the joint. It is this decentering and imbalance of the shoulder that will be responsible for subacromial impingement.

Symptoms and manifestations

Subacromial impingement pain

Pain

Pain is the main symptom of subacromial impingement. It is most often inflammatory pain, which will be nocturnal and may cause insomnia.

Pain may also cause nocturnal awakenings especially when turning on the affected shoulder. Sometimes pain is rather morning with unlocking phenomenon.

Triggering phenomenon: It is the arm elevation movements that will stimulate or trigger painful phenomena, often from 80° or 90° with sensation of catching or audible cracking.

Functional impairment

Functional impairment

Functional impairment corresponds to an inability to normally mobilize the affected limb. In the context of subacromial impingement, it is most often a functional discomfort rather than true impairment.

A temporary and transient picture of paralyzed shoulder or "dead arm" may also appear, with total inability to actively mobilize the shoulder. This situation most often occurs after prolonged activity with the arm in the air.

Lack of strength: In some cases, functional impairment will manifest as a lack of strength corresponding to difficulty or impossibility of performing movements against resistance.

Diagnosis and evaluation

Clinical examination

The clinical examination will find signs of subacromial impingement more or less marked. Several clinical tests will allow to detect signs in favor of subacromial impingement:

  • Neer maneuver
  • Yocum test
  • Hawkins test
  • Gerber test

Typically, passive shoulder mobilization should be preserved even if active mobilization is limited or painful.

Imaging examinations

Conflict X-ray

X-ray

Routine examination to evaluate bone structures and visualize acromial shape (Bigliani)

Conflict ultrasound

Ultrasound

Minimally invasive examination to explore tendon structures and look for signs of bursitis

Conflict arthro-CT

Arthro-CT/MRI

Reference examination to specify conflict and associated lesions (transfixing or not)

Therapeutic options

Conservative treatment

Conservative conflict treatment

Treatment choice is made case by case and will depend on many clinical and paraclinical factors. Duration of symptom evolution, importance of clinical manifestations, response to different previously undertaken treatments are elements that will be taken into account.

  • Drug treatment: Level 1-2 analgesics, anti-inflammatories
  • Immobilization: Orthopedic vest maximum 1 week to 10 days
  • Cryotherapy: Multiple daily cold applications
  • Cortisone infiltration: Injection into subacromial space
  • Physiotherapy: Fundamental role for lifting subacromial impingement

Surgical treatment

Surgical treatment consists of performing an acromioplasty. This acromioplasty corresponds to decompression of the subacromial space by filing and thinning of the acromial bone at its deep part.

Surgical indications:

  • Prolonged or recurrent subacromial impingement
  • Ineffectiveness of well-conducted symptomatic treatment
  • Ineffectiveness of well-followed physiotherapy
  • Aggressive nature of acromial vault
Arthroscopic acromioplasty

Arthroscopic acromioplasty

Intervention performed under arthroscopy allowing to file and sand the deep part of the acromion. Arthroscopy corresponds to the introduction of a camera into the joint and subacromial space to explore and treat different lesions.

Advantages: Absence of muscle lesion, lower risk of infection or hemorrhage, quasi absence of post-operative scar at distance.

Rehabilitation and physiotherapy

Subacromial impingement rehabilitation

Physiotherapy has a fundamental role in the background treatment of subacromial impingement, whether there has been surgical intervention or not. Chronic shoulder pain tends to create diffuse muscle contractures of peri-scapular muscles.

Physiotherapy principles:

  • Massages: Muscle relaxation to avoid reactive contractures
  • Cryotherapy: Analgesic and anti-inflammatory role
  • Mobilization: Work of decompression and stretching of subacromial space
  • Recentering: Work of shoulder recentering and decompression

Self-physiotherapy:

Self physiotherapy corresponds to all active or passive mobilization activities that the patient will be able to perform by himself. The goal is not to substitute for classic physiotherapy sessions but it is a fundamental complement.

Principles: Work without pain, very regular work (5 minutes 5x per day), work guided by the physiotherapist.

Possible complications

Infection

Hemorrhage/hematoma

Nerve lesion

Painful recurrence

Tendinitis/bursitis

Complex regional pain syndrome

Practical information

Hospitalization

0 to 2 days (outpatient)

Anesthesia

General

Immobilization

15 days to 1 month

Physiotherapy

2 to 3 months

Control

1 month